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K., Tang, Y., Alvarez-Buylla, A

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However, its application in long-term implantable devices is often limited by issues such as uncontrolled enzymatic degradation, insufficient mechanical strength, and rapid dissolution in physiological environments 2

reported that VHL loss increases intracellular lactate to drive H3K18la-dependent activation of the PDGFRB promoter, forming a feed-forward loop in which PDGFR signaling reinforces glycolysis and histone lactylation